Straight forceps (Birkitt's tonsil artery forceps) used to catch the bleeding point. (in tonsillar fossa) following tonsillectomy.
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| Birkitt's first tonsillar artery forceps |
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| Wilson's second artery forceps |
Thats all
- Jaskunwar Singh
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| Birkitt's first tonsillar artery forceps |
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| Wilson's second artery forceps |
Hey!
I'm clearly doing a lot of notes + mnemonic posts. Here's another one on Langerhans cell histiocytosis!
Langerhans lingers
L - Lytic lesion of bones
I - Diabetes Insipidus
N - Nil
G - Eosinophilic Granulomas
E - Exophthalmos
R - Rrrrr.... :P
S - Skin rash
From study group:
Any mnemonic for Hand Schuller Christian disease?
Mnemonic by IkaN: Hands LIE
Lytic bone lesions
Insipidus (Diabetes Insipidus)
Exophthalmos
Mnemonic by Dev: CDE
Calavarial skull defect
Diabetes insipidus
Exophthalmos
Disclaimer: It's a morbid mnemonic. If you are sensitive and religious, don't read it.
Mnemonic by Vinayak: Jesus CHRIST was killed by hammering nails into his HAND, he DIed, his hands' bone got LYSED and we celebrate EXmas:
For calvarial skull defects, you know, wood was also hammered into his head. DI is diabetes insipidus. EX is exophthalmos.
That's all!
-IkaN
So basically, ECG is an electrical representation of heart activity on ECG paper.
Here are some important postulates of ECG paper:
The needle of galvanometer travels 300 big squares in 1 minute or 60 seconds.
That makes 0.2 seconds for each big square.
In turn each big square is divided into 5 small boxes /squares.
So we get each small box counting about
0.04 seconds.
So let's review all calculations:
1 Big square = 0.2 seconds.
1 small square = 0.04 seconds.
Each P wave travels about 2 and 1/2 small squares making upto 0.1 second.
The segment PQ (Sometimes we use PR) again takes 2 and 1/2 small squares making upto 0.1 second.
QRS complex used another 2 and 1/2 small squares making another 0.1 second.
While S-T segments uses 10 small squares forming using 0.4 seconds.
Note: We normally consider 2 and 1/2 small squares but there can be 2 or 3 small squares.
~Ojas
Here's another notes + mnemonic post on Friedreichs Ataxia
Genetic stuff:
- Due to mutated frataxin gene
- GAA trinucleotide repeats
- Autosomal recessive
- Mitochondrial iron accumulation due to loss of iron transporter coded by frataxin gene results in oxidized intramitochondrial iron accumulation.
Clinical stuff:
- Cardiomyopathy is the most common cause of death in FA. Cardiomegaly, conduction defects, murmurs, fibrosis are seen.
- Musculoskeletal abnormalities like foot deformities (Hammer toe, pes cavus, pes equinovarus) and scoliosis is seen.
- Diabetes mellitus is seen in 20% of patients
Neurologic stuff:
- Nystagmus
- Titubation
- Dysarthria
- Dysmetria
- Ataxia of trunk and limb movements.
- Loss of proprioception and vibratory sense.
- Extensor plantar response, absence of deep tendon reflexes and weakness is found.
That's all!
Stuff.
-IkaN
Mnemonic: Friedreichs FRIEDS!
Duodenal atresia
Hey!
In this post, I wanted to emphasize duodenal atresia is associated with double bubble sign.
It's common in Downs syndrome and polyhydramnios maybe seen in utero.
Bilious vomiting is seen because the obstruction is after the ampulla of Vater.
Because the child is vomiting, abdominal distension is not seen.
That's all!
I need sleep right now.
-IkaN