Uses: Examination of external ear and tympanic membrane
In operative procedures like myringoplasty, myringotomy.
Removal of wax, foreign body, otomycosis, ear discharge.
Different sizes for patients according to age.
Thats all
- Jaskunwar Singh
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| Birkitt's first tonsillar artery forceps |
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| Wilson's second artery forceps |
Hey!
I'm clearly doing a lot of notes + mnemonic posts. Here's another one on Langerhans cell histiocytosis!
Langerhans lingers
L - Lytic lesion of bones
I - Diabetes Insipidus
N - Nil
G - Eosinophilic Granulomas
E - Exophthalmos
R - Rrrrr.... :P
S - Skin rash
From study group:
Any mnemonic for Hand Schuller Christian disease?
Mnemonic by IkaN: Hands LIE
Lytic bone lesions
Insipidus (Diabetes Insipidus)
Exophthalmos
Mnemonic by Dev: CDE
Calavarial skull defect
Diabetes insipidus
Exophthalmos
Disclaimer: It's a morbid mnemonic. If you are sensitive and religious, don't read it.
Mnemonic by Vinayak: Jesus CHRIST was killed by hammering nails into his HAND, he DIed, his hands' bone got LYSED and we celebrate EXmas:
For calvarial skull defects, you know, wood was also hammered into his head. DI is diabetes insipidus. EX is exophthalmos.
That's all!
-IkaN
So basically, ECG is an electrical representation of heart activity on ECG paper.
Here are some important postulates of ECG paper:
The needle of galvanometer travels 300 big squares in 1 minute or 60 seconds.
That makes 0.2 seconds for each big square.
In turn each big square is divided into 5 small boxes /squares.
So we get each small box counting about
0.04 seconds.
So let's review all calculations:
1 Big square = 0.2 seconds.
1 small square = 0.04 seconds.
Each P wave travels about 2 and 1/2 small squares making upto 0.1 second.
The segment PQ (Sometimes we use PR) again takes 2 and 1/2 small squares making upto 0.1 second.
QRS complex used another 2 and 1/2 small squares making another 0.1 second.
While S-T segments uses 10 small squares forming using 0.4 seconds.
Note: We normally consider 2 and 1/2 small squares but there can be 2 or 3 small squares.
~Ojas
Here's another notes + mnemonic post on Friedreichs Ataxia
Genetic stuff:
- Due to mutated frataxin gene
- GAA trinucleotide repeats
- Autosomal recessive
- Mitochondrial iron accumulation due to loss of iron transporter coded by frataxin gene results in oxidized intramitochondrial iron accumulation.
Clinical stuff:
- Cardiomyopathy is the most common cause of death in FA. Cardiomegaly, conduction defects, murmurs, fibrosis are seen.
- Musculoskeletal abnormalities like foot deformities (Hammer toe, pes cavus, pes equinovarus) and scoliosis is seen.
- Diabetes mellitus is seen in 20% of patients
Neurologic stuff:
- Nystagmus
- Titubation
- Dysarthria
- Dysmetria
- Ataxia of trunk and limb movements.
- Loss of proprioception and vibratory sense.
- Extensor plantar response, absence of deep tendon reflexes and weakness is found.
That's all!
Stuff.
-IkaN
Mnemonic: Friedreichs FRIEDS!