And here's the answer for it.
#Instruments
B. Adenoidectomy
C. Tonsillectomy
D. Parathyroidectomy
Thank you.
Let us suppose that a patient has the classic signs & symptoms of Asthma and with that she has eosinophilia ( Absolute eosinophil count above 500/mm3).
The next step should be to look at the serum IgE levels.
If it is also elevated-
1. Do a stool examination and serological testing for strongyloidiasis.
2. Do Aspergillus-skin testing and check for aspergillus specific IgE.
If you have got a positive result for one of the above two, you have your diagnosis.
But what if both of the above results come out to be negative.
Then consider Churg Strauss syndrome or Eosinophilic granulomatosis with polyangiitis.
That's it!
-VM
Hey guys!
I saw a patient today, a 37 year old female patient with chief complaints of cough and shortness of breath. She has a history of allergic rhinitis and migraine. She is also obese with an BMI of 31.
As you must have guessed already she was diagnosed with Bronchial asthma. On the spirometry report done 4 months back, there was an obstructive pattern and after giving bronchodilators her FEV1 increased by 22% (>12%) and FEV1 vol increased by 300ml (>200ml). These findings also strongly support the diagnosis of asthma.
She was started on Albuterol as needed and Salmeterol-fluticasone MDI. She didn't show good response in the first 4 weeks, so she was also started on Montelukast and Tiotropium inhaler.
Now after 4 months, she still had cough and shortness of breath. She had bilateral polyphonic wheezes. A chest X Ray was done which came out to be normal. On pulse oximetry, SaO2 was 97% while breathing ambient air. On chest CT we found two attributes:
Subpleural opacities and Ground glass opacities.
So based on the CT scan findings, differential diagnosis:
Subpleural opacities:
1. Eosinophilic granulomatosis with polyangiitis( previously called Churg Strauss).
2. Organizing pneumonia
3. Pulmonary embolism with resultant subpleural pulmonary infarction
4. Allergic bronchopulmonary aspergillosis ( well, not really, actually in this case, there is peripheral air space opacification which looks identical to subpleural opacities)
Now Ground glass opacities:
1. Atypical pneumonia
2. Hypersensitivity pneumonia
3. Several ILDs
4. Sarcoidosis
5. Pulm Edema
6. Pulm Haemorrhage
That's all!
-VM
Hello awesomites!
Here's a collection of Cutaneous infections/diseases caused by Streptococcus pyogenes
Direct infections of skin or subcutaneous tissue-
1. Cellulitis
2. Impetigo
3.Ecthyma,Erysipelas
4. Vulvovaginitis
5. Perianal infection
6. Streptococcal ulcers
7. Blistering distal dactylitis
8. Necrotizing fasciitis
Secondary infection-
Eczema, infestations, ulcers, etc.
Tissue damage from circulating toxins-
1. Scarlet fever
2. Toxic‐shock‐like syndrome
3. Recurrent toxin‐mediated perineal erythema
Skin lesions due to allergic hypersensitivity to Streptococcal antigens
1. Erythema nodosum
2. Vasculitis
Skin disease provoked or influenced by Streptococcal infection (mechanism uncertain)-
1. Psoriasis, especially guttate forms.
2. Kawasaki disease.
That's all!
MD Mobarak Hussain (Maahii)
The optic disc margins start to blur in the following order in papilledema-
1. Superior
2. Inferior
3. Nasal
4. Temporal
Blurring first occurs where the nerve fibre density is the highest.The optic disc being ovoid in shape, with taperings superiorly and inferiorly, the nerve fibre density is higher there. Hence such a sequence.
That's all!
-Sushrut Dongargaonkar